Introduction
Eating
disorders refer to a group of conditions defined
by abnormal eating
habits that may involve either insufficient or excessive food
intake to the detriment of an individual's physical
and mental
health. Bulimia
nervosa, anorexia
nervosa, and binge eating disorder
are the most common specific forms in the United States. Though primarily
thought of as affecting females (an estimated 5–10 million being affected
in the U.S.), eating disorders affect males as well (an estimated
1 million U.S. males being affected). Although eating disorders are
increasing all over the world among both men and women, there is evidence to
suggest that it is women in the Western world who are at the highest risk of
developing them and the degree of westernization
increases the risk.2
Bulimia nervosa, in many ways, represents a failed attempt at anorexia
nervosa, sharing the goal of becoming very thin, but occurring in an individual
less able to sustain prolonged semi-starvation or severe hunger as consistently
as classic restricting anorexia nervosa patients. These eating binges provoke
panic as individuals feel that their eating has been out of control. The
unwanted binges lead to secondary attempts to avoid the feared weight gain by a
variety of compensatory behaviors, such as purging or excessive exercise.2
DEFNITION.
In the text revision of the 4th edition of
Diagnostic and Statistical Manual of Mental Disorders (DSM-IV-TR), bulimia
nervosa is defined as binge eating combined with inappropriate ways of stopping
weight gain. Social interruption or physical discomfort that is, abdominal pain
or nausea terminates the binge eating, which is often followed by feelings of
guilt, depression, or self-disgust. Unlike patients with anorexia nervosa,
those with bulimia nervosa may maintain a normal body weight.3
EPIDEMIOLOGY
Bulimia nervosa is more prevalent than anorexia
nervosa. Estimates of bulimia nervosa range from 2 to 4 percent of young women.
As with anorexia nervosa, bulimia nervosa is significantly more common in women
than in men, but its onset is often later in adolescence than that of anorexia
nervosa. According to DSM-IV-TR, the rate of occurrence in males is one tenth
of that in females. The onset may even occur in early adulthood. Approximately
20 percent of college women experience transient bulimic symptoms at some point
during their college years. Although bulimia nervosa is often present in
normal-weight young women, they sometimes have a history of obesity.3
History
Russell described bulimia nervosa in 1979, using
the term nervosa to unite the two forms of the eating disorders spectrum—the
self-starving syndrome of anorexia nervosa and the newly recognized binge–purge
disorder—psycho pathologically. Bulimia nervosa was initially described as “an
ominous variant of anorexia nervosa,” but later descriptions incorporated the
syndrome of bulimia nervosa at normal weight.
The recognition of bulimia nervosa was hidden for a
number of reasons, including the shame and secrecy of sufferers, who were
reluctant to reveal these symptoms even while being treated for other related
comorbid disorders, such as depression; the seemingly normal weight of most
bulimic patients; and the lack of requests for help. The “night-eating binge
syndrome,” characterized by often unremembered binge eating while still in some
stage of sleep, is even more difficult to diagnose, but increasingly is
recognized as a true variant. Despite the relatively recent formal description
of bulimia nervosa, evidence-based treatment strategies have emerged quickly
and have proven more effective than those for anorexia nervosa.3
The recent history of eating disorders has focused
on several areas, including (1) better delineation of the non compensating
binge-eating disorder, (2) clearer descriptions of the prevalence and syndrome
characteristics of eating disorders in men, and (3) studies recognizing the
contributions of genetic and other predisposing neurobiological factors
interacting with socio cultural norms..
By the late
20th century, fatty, palatable foods were available to most populations in
Western countries, and, paradoxically, high-fat foods became particularly cheap
and available for many low-income populations. Obesity has become much more
prominent among all social classes, particularly poorer populations.
Concurrently, pressures to be thin, weight control efforts, and eating
disorders as clinical problems have become increasingly associated with
affluent societies.3
ETIOLOGY
Biological Factors
Some investigators have attempted to associate
cycles of binging and purging with various neurotransmitters. Because
antidepressants often benefit patients with bulimia nervosa and because
serotonin has been linked to satiety, serotonin and norepinephrine have been
implicated. Because plasma endorphin levels are raised in some bulimia nervosa
patients who vomit, the feeling of well-being after vomiting that some of these
patients experience may be mediated by raised endorphin levels. According to
DSM-IV-TR, increased frequency of bulimia nervosa is found in first-degree
relatives of persons with the disorder.4
Social Factors
Patients with bulimia nervosa, as with those with
anorexia nervosa, tend to be high achievers and to respond to societal
pressures to be slender. As with anorexia nervosa patients, many patients with
bulimia nervosa are depressed and have increased familial depression, but the
families of patients with bulimia nervosa are generally less close and more
conflictual than the families of those with anorexia nervosa. Patients with
bulimia nervosa describe their parents as neglectful and rejecting.4
Psychological Factors
Patients with bulimia nervosa, as with those with
anorexia nervosa, have difficulties with
adolescent demands, but patients with bulimia nervosa are more outgoing, angry, and impulsive than those with anorexia nervosa. Alcohol dependence, shoplifting, and emotional
adolescent demands, but patients with bulimia nervosa are more outgoing, angry, and impulsive than those with anorexia nervosa. Alcohol dependence, shoplifting, and emotional
lability (including suicide attempts) are
associated with bulimia nervosa. These patients generally experience their
uncontrolled eating as more ego-dystonic than do patients with anorexia nervosa
and so seek help more readily.
Patients with bulimia nervosa lack superego control
and the ego strength of their counterparts with anorexia nervosa. Their
difficulties controlling their impulses are often manifested by substance
dependence and self-destructive sexual relationships in addition to the binge
eating and purging that characterize the disorder. Many patients with bulimia
nervosa have histories of difficulties separating from caretakers, as
manifested by the absence of transitional objects during their early childhood
years. Some clinicians have observed that patients with bulimia nervosa use
their own bodies as transitional objects. The struggle for separation from a
maternal figure is played out in the ambivalence toward food; eating may
represent a wish to fuse with the caretaker, and regurgitating may
unconsciously express a wish for separation. 4
ICD 10 CLASSIFICATION
A. There are recurrent episodes
of overeating (at least twice a week over a period of 3 months) in which
large amounts of food are consumed in short periods.
B. There is persistent
preoccupation with eating and a strong desire or a sense of compulsion to eat
(craving).
C. The patient attempts to
counteract the “fattening” effects of food by one or more of the following:
(1)
self-induced vomiting
(2)
self-induced purging
(3)
alternating periods of starvation
(4) use of
drugs such as appetite suppressants, thyroid preparations, or diuretics; when
bulimia occurs in diabetic patients, they may choose to neglect their insulin
treatment
D. There is self-perception of
being too fat, with an intrusive dread of fatness (usually leading to
underweight).
Atypical bulimia nervosa
Researchers studying atypical
forms of bulimia nervosa, such as those involving normal or excessive body
weight, are recommended to make their own decisions about the number and type
of criteria to be fulfilled.2
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Pathology
and Laboratory Examinations
Bulimia nervosa can result in electrolyte
abnormalities and various degrees of starvation, although it may not be as
obvious as in low-weight patients with anorexia nervosa. Thus, even
normal-weight patients with bulimia nervosa should have laboratory studies of
electrolytes and metabolism. In general, thyroid function remains intact in
bulimia nervosa, but patients may show nonsuppression on the
dexamethasone-suppression test. Dehydration and electrolyte disturbances are
likely to occur in patients with bulimia nervosa who purge regularly. These
patients commonly exhibit hypomagnesemia and hyperamylasemia. Although not a
core diagnostic feature, many patients with bulimia nervosa have menstrual
disturbances. Hypotension and bradycardia occur in some patients.4
DIFFERENTIAL
DIAGNOSIS
The diagnosis of bulimia nervosa cannot be made if
the binge-eating and purging behaviors occur exclusively during episodes of
anorexia nervosa. In such cases, the diagnosis is anorexia nervosa, binge eating-purging
type.
Clinicians must ascertain that patients have no
neurological disease, such as epileptic-equivalent seizures, central nervous
system tumors. The pathological features manifested by Klüver-Bucy syndrome
are visual agnosia, compulsive licking and biting, examination of objects by
the mouth, inability to ignore any stimulus, placidity, altered sexual behavior
(hypersexuality), and altered dietary habits, especially hyperphagia. The
syndrome is exceedingly rare and is unlikely to cause a problem in differential
diagnosis. Kleine-Levin syndrome consists of periodic hypersomnia lasting for 2
to 3 weeks and hyperphagia. As in bulimia nervosa, the onset is usually during
adolescence, but the syndrome is more common in men than in women.
Patients
with bulimia nervosa who have concurrent seasonal affective disorder and
patterns of atypical depression (with overeating and oversleeping in low-light
months) may manifest seasonal worsening of both bulimia nervosa and depressive
features. In these cases, binges are typically much more severe during winter
months. Bright light therapy (10,000 lux for 30 minutes, in early morning, at
18 to 22 inches from the eyes) may be a useful component of comprehensive
treatment of an eating disorder with seasonal affective disorder.
Some patients with bulimia nervosa perhaps 15
percent have multiple comorbid impulsive behaviors, including substance abuse,
and lack of ability to control themselves in such diverse areas as money
management (resulting in impulse buying and compulsive shopping) and sexual
relationships (often resulting in brief, passionate attachments and
promiscuity). They exhibit self-mutilation, chaotic emotions, and chaotic
sleeping patterns. They often meet criteria for borderline personality disorder
and other mixed personality disorders and, not infrequently, bipolar II
disorder.
COURSE
AND PROGNOSIS
Bulimia nervosa is characterized by higher rates of
partial and full recovery compared with anorexia nervosa. As noted in the
treatment section, those treated fare much better than those untreated.
Patients untreated tend to remain chronic or may show small, but generally
unimpressive degrees of improvement with time. In a 10-year follow-up study of
patients who had previously participated in treatment programs, the number of
women who continued to meet full criteria for bulimia nervosa decline
as the duration of follow-up increased.
Approximately 30 percent continued to engage in recurrent binge-eating or
purging behaviors. A history of substance use problems and a longer duration of
the disorder at presentation predicted worse outcome. Depending on definitions,
38 to 47 percent of women were fully recovered at follow-up.4
TREATMENT
Most patients with uncomplicated bulimia nervosa do
not require hospitalization. In general, patients with bulimia nervosa are not
as secretive about their symptoms as patients with anorexia nervosa. Therefore,
outpatient treatment is usually not difficult, but psychotherapy is frequently
stormy and may be prolonged. Some obese patients with bulimia nervosa who have
had prolonged psychotherapy do surprisingly well. In some cases—when eating
binges are out of control, outpatient treatment does not work, or a patient
exhibits such additional psychiatric symptoms as suicidality and substance
abuse—hospitalization may become necessary. In addition, electrolyte and
metabolic disturbances resulting from severe purging may necessitate
hospitalization.2
Psychotherapy
Cognitive-Behavioral Therapy
Cognitive-behavioral therapy (CBT) should be
considered the benchmark, first-line treatment for bulimia nervosa. The data
supporting the efficacy of CBT are based on strict adherence to rigorously
implemented, highly detailed, manual-guided treatments that include about 18 to
20 sessions over 5 to 6 months. CBT implements a number of cognitive and
behavioral procedures to (1) interrupt the self-maintaining behavioral cycle of
bingeing and dieting and (2) alter the individual's dysfunctional cognitions;
beliefs about food, weight, body image; and overall self-concept.2
Dynamic Psychotherapy
Psychodynamic treatment of patients with bulimia
nervosa has revealed a tendency to concretize introjective and projective
defense mechanisms. In a manner analogous to splitting, patients divide food
into two categories: items that are nutritious and those that are unhealthy.
Food that is designated nutritious may be ingested and retained because it
unconsciously symbolizes good introjects. But junk food is unconsciously
associated with bad introjects and, therefore, is expelled by vomiting, with
the unconscious fantasy that all destructiveness, hate, and badness are being
evacuated. Patients can temporarily feel good after vomiting because of the
fantasized evacuation, but the associated feeling of âll good is short-lived
because it is based on an unstable combination of splitting and projection.4
Pharmacotherapy
Antidepressant medications have
been shown to be helpful in treating bulimia. This includes the selective
serotonin reuptake inhibitors (SSRIs), such as fluoxetine. This may be based on
elevating central 5-hydroxytryptamine levels. Antidepressant medications can
reduce binge eating and purging independent of the presence of a mood disorder.
Thus, antidepressants have been used successfully for particularly difficult
binge-purge cycles that do not respond to psychotherapy alone. Imipramine
(Tofranil), desipramine (Norpramin), trazodone (Desyrel), and monoamine oxidase
inhibitors (MAOIs) have been helpful. In general, most of the antidepressants
have been effective at dosages usually given in the treatment of depressive
disorders. Dosages of fluoxetine that are effective in decreasing binge eating,
however, may be higher (60 to 80 mg a day) than those used for depressive
disorders. Medication is helpful in patients with comorbid depressive disorders
and bulimia nervosa. Carbamazepine (Tegretol) and lithium (Eskalith) have not
shown impressive results as treatments for binge eating, but they have been
used in the treatment of patients with bulimia nervosa with comorbid mood
disorders, such as bipolar I disorder.1
Nursing
Intervention.
IMBALANCED
NUTRITION:MORE THAN BODY REQUIRMENT related
to
[Compulsive eating]
Excessive intake in relation to
metabolic needs
[Sedentary lifestyle]
[Genetics]
[Unmet dependency needs—fixation
in oral developmental stage]evidenced
Weight 20 percent over ideal for
height and frame
[Body mass index of 30 or more]
Goals/Objectives
Short-Term
Goal
Client will verbalize
understanding of what must be done to lose weight.
Long-Term
Goal
Client will demonstrate change in
eating patterns resulting in a steady weight loss.
Interventions with Selected Rationales
1. Encourage the client to keep a
diary of food intake. A food diary
provides the opportunity for the client to gain a realistic picture of the
amount of food ingested and provides data on which to base the dietary program.
2. Discuss feelings and emotions
associated with eating. This helps
to identify when client is eating to satisfy an emotional need rather than a
physiological one.
3. With input from the client,
formulate an eating plan that includes food from the basic food pyramid with
emphasis on low-fat intake. It is helpful to keep the plan as similar to the
client’s usual eating pattern as possible. Diet must eliminate calories while maintaining adequate nutrition. Client is
more likely to stay on the
eating plan if he or she is able to participate in its creation and it deviates as little as possible from usual
types of foods.
4. Identify realistic increment
goals for weekly weight loss. Reasonable
weight loss (1 to 2 lb/wk) results in more lasting effects. Excessive, rapid
weight loss may result in fatigue and irritability and may ultimately lead to
failure in meeting goals for weight loss. Motivation is more easily sustained
by meeting “stair-step” goals.
5. Plan progressive exercise
program tailored to individual goals and choice. Exercise may enhance weight loss by burning calories and reducing appetite, increasing energy, toning
muscles, and enhancing sense
of well-being and accomplishment. Walking is an excellent choice for overweight individuals.
6. Discuss the probability of
reaching plateaus when weight remains stable for extended periods. Client should know that this is likely
to happen as changes in metabolism
occur. Plateaus cause frustration, and client may need additional support
during these times to remain
on the weight loss program.
7. Provide instruction about
medications to assist with weight loss if ordered by the physician. Appetite-suppressant drugs (e.g.,
sibutramine)may be helpful to someone who is obese. Drugs should be used for this purpose for only a short
period while the individual attempts to adjust to the new pattern of eating.
Outcome Criteria
1. Client has established a
healthy pattern of eating for weight control
the weight loss progressing toward a desired goal.
2. Client verbalizes plans for
future maintenance of weight control.
● DISTURBED BODY IMAGE/LOW
SELF-ESTEEM related to
[Dissatisfaction with appearance]
[Unmet dependency needs]
[Lack of adequate nurturing by
maternal figure]
evidenced by
Negative feelings about body
(e.g., feelings of helplessness, hopelessness, or powerlessness)
[Verbalization of desire to lose
weight]
[Failure to take responsibility
for self-care (self-neglect)]
Lack of eye contact
[Expressions of low self-worth]
Goals/Objectives
Short-Term
Goal
Client will begin to accept self
based on self-attributes rather than on appearance.
Long-Term
Goal
Client will pursue loss of weight
as desired.
Interventions with Selected Rationales
1. Assess client’s feelings and
attitudes about being obese. Obesity
and compulsive eating behaviors may have deep-rooted psychological
implications, such as compensation for lack of love and nurturing or a defense
against intimacy.
2. Ensure that the client has
privacy during self-care activities. The
obese individual may be sensitive or self-conscious about his or her body.
3. Have client recall coping
patterns related to food in family of origin, and explore how these may affect
current situation. Parents
are role models for their children.
Maladaptive eating behaviours are learned within the family system and are supported through positive reinforcement. Food may be substituted
by the parent for
affection and love, and eating is associated with a feeling of satisfaction, becoming the primary
defense.
4. Determine client’s motivation
for weight loss and set goals. The
individual may harbour repressed feelings of hostility, which may be expressed
inward on the self. Because of a poor self-concept, the person often has
difficulty with relationships. When the motivation is to lose weight for
someone else, successful weight loss is less likely to occur.
5. Help client identify positive
self-attributes. Focus on strengths and past accomplishments unrelated to
physical appearance. It is
important that self-esteem not be
tied solely to size of the body.Client needs to recognize that obesity need not
interfere with positive feelings
regarding self-concept and self-worth
6. Refer client to support or
therapy group. Support groups can
provide companionship, increase motivation, decrease loneliness and social
ostracism, and give practical solutions to common problems.
Group
therapy can be helpful in dealing with underlying psychological concerns.5
. CONCLUSION:
Prevention
will only be possible when specific and reversible factors that lead to the
initiation of eating disorders have been identified. However, treatment is
often provided on an empirical basis. Here, the most pressing need is an
intensive and comprehensive inpatient weight restoration programme for patients
with bulimia nervosa against outpatient
supportive care plus access to brief hospitalization for nutritional
stabilization.
Discussion As a health care professional what type of
preventive measures can apply to the public
REFERENCES:
- Boyd MA .Psychiatric Nursing Contemporary Practice 2nd edition. Philadelphia: Lippincott Publications; 2001.
- Kaplan HI, Saddock BJ. Synopsis of Psychiatry, Behavioral Sciences/ Clinical Psychiatry .9th edition. Hong Kong: William and Wilkinson Publishers; 1998.
- Schultz JM.Videback SL.Lippincott’s Manual of Psychiatric Nursing care Plans.6th edition. Philadelphia: Lippincott Williams & Wilkins; 2002.
- Stuart GW, Laria MT. Principles and Practices of Psychiatric Nursing. Philadelphia: Mosby Publishers; 2001.
- Townsend M C Psychiatric Mental Health Nursing- concepts of care. 5th edition. Philadelphia: F.A Dais Company; 2005.
- http://www.ncbi.nlm.nih.gov/pubmedhealth/PMH0001401/
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